1. Light Contact
The glans (head) of the penis is composed of soft, highly vascular erectile tissue with a mucosal surface.
When the two glans touch, the initial sensation would be mild pressure on the sensitive distal tissue.
Because the surface is smooth and moist, the contact would be slightly adhesive—more like soft tissue pressing on soft tissue, not rigid surfaces.
2. Mild Pressure
As pressure increases, the skin and subcutaneous tissues begin to compress.
The glans flatten slightly against each other, distributing the load across their broad surfaces.
Blood within the erectile tissue momentarily redistributes, making the tissue slightly firmer but also more tender.
3. Moderate Pressure
Compression now begins to exceed the natural pliability of the glans.
The following effects emerge:
Blanching of the compressed regions due to reduced capillary blood flow.
Increased friction because soft tissues are being displaced laterally.
Localized discomfort as mechanoreceptors and nociceptors respond to deforming pressure.
The urethral opening (meatus) of each penis is pressed off-center or slightly distorted.
4. High Pressure
At this stage, the tissue response becomes visibly strained.
Likely effects:
Reddening around the edges where blood is forced outward.
Edema (brief swelling) due to fluid displacement and microvascular congestion.
Pain becomes sharp rather than dull.
The structural limitation is not the erectile cylinders but the glans tissue itself, which is not engineered to withstand shearing or compression loads from another glans.
5. Very High Pressure
Now the force exceeds the comfortable tolerance of soft erectile tissue.
Potential outcomes:
Surface microtrauma: tiny ruptures in superficial capillaries leading to immediate bruising.
Shearing stress: the glans may shift relative to the underlying erectile structure, producing a burning or tearing sensation.
Instability: the penises may slip to one side because the smooth mucosal surfaces cannot maintain alignment under intense force.
6. Extreme Pressure
The glans cannot act as load-bearing structures, so they deform significantly.
Likely physiological consequences:
Significant bruising in the compressed regions.
Possible small hematomas (localized bleeding under the surface).
Acute pain due to overstimulation of nociceptors.
If the force is truly extreme and sustained, risk of laceration or abrasions emerges—particularly if lateral movement is introduced.
7. Absolute Maximum Force Before Structural Injury
The erectile bodies beneath the glans may partially deflate reflexively due to protective autonomic responses.
Pain response peaks.
Structural injury becomes possible:
Tearing of mucosal tissue
Rupture of superficial blood vessels
Abrasion from forced friction
At no stage does any meaningful “interface” form between the two structures; increasing pressure simply increases deformation and discomfort.
1. Light contact (0–5 N)
Glans-to-glans touch. Soft, warm, slightly compressive. Urethral openings align or kiss. Skin slides a little on the underlying corpora spongiosa. Mild indentation, no real deformation yet. Both remain fully erect.
2. Moderate pressure (5–20 N)
The glans begin to flatten against each other like two firm mushrooms being squashed. The coronal ridges compress and splay outward. The urethral meatuses are forced shut or everted slightly. Blood is displaced toward the shaft; the heads turn a deeper red/purple. Still no pain, just a dull, full sensation.
3. Firm pressure (20–50 N)
Significant flattening. The glans now resemble soft hockey pucks pressed face-to-face. The corpora cavernosa start to bow inward under axial load; you can feel the rigid tunica albuginea resisting. Veins blanch on the surface. A faint ache appears deep in the pelvis as the suspensory ligaments and crura take load.
4. Hard pressure (50–100 N)
Full pancake stage. Each glans is roughly 50–60 % of its original depth. The frenula are stretched flat. Urethral openings are completely occluded and may prolapse outward like tiny pink donuts. Shafts begin to buckle slightly at the base despite erection; the tunica is now the only thing preventing rupture. Pain shifts from ache to sharp, burning.
5. Extreme pressure (100–200+ N)
Structural limits approached. The tunica albuginea (the fibrous sheath) reaches its tensile limit (~200–300 N in cadaver studies before rupture). One or both penises will start to bend laterally or fold in the middle. Possible outcomes in ascending order of probability:
Corporal fracture: loud snap, immediate detumescence, eggplant deformity, massive hematoma.
Rupture of the urethra or corpus spongiosum: blood from meatus.
Bilateral buckling without fracture: both shafts fold like bent steel rods, extreme pain, possible vascular tearing.
6. Beyond humanly sustainable force
Total mechanical failure. Tunica tears open, corpora cavernosa extrude like split sausage casing, torrential bleeding, irreversible deformity, emergency surgery territory.
In short: starts as warm mushy docking, ends (if you somehow keep pushing) with two exploded eggplants and a trip to the ER.
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